Review ArticleReview
Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode
Spiros A. Vlahopoulos
Cancer Biology & Medicine August 2017, 14 (3) 254-270; DOI: https://doi.org/10.20892/j.issn.2095-3941.2017.0029
Spiros A. Vlahopoulos
1The First Department of Pediatrics, University of Athens, Horemeio Research Laboratory, Athens 11527, Greece

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In this issue
Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode
Spiros A. Vlahopoulos
Cancer Biology & Medicine Aug 2017, 14 (3) 254-270; DOI: 10.20892/j.issn.2095-3941.2017.0029
Jump to section
- Article
- Abstract
- Introduction
- Cycle of NF-κB activity
- Molecular types induced by NF-κB
- Operating mode of NF-κB
- Loss of feedback to NF-κB affects tissue homeostatic coordination
- Interacting agents of NF-κB direct immune system activity
- Intracellular and extracellular cancer processes
- Modulation of the microenvironment to “license” cancer: many processes initiated by a few genes
- NF-κB target gene regulation is tissue dependent
- Conclusions
- Acknowledgements
- Footnotes
- References
- Figures & Data
- Info & Metrics
- References
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